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doi: 10.1074/jbc.M114.625053

41 As discussed above, mitochondrial dysfunction (mitochondrial bioenergetics, calcium buffering) and mitochondrial quality control systems (mitochondrial dynamics, mitophagy, mitochondrial biogenesis) may be involved in the pathological mechanisms of diabetes-related NDs

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As some neuronal responsiveness was preserved after purinergic blockade, however, purinergic receptors insensitive to PPADS, or other molecules secreted from GLUTag cells may participate in the communication with ND ganglion neurons in co-culture

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